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They not only identified Rho A as a target of the human epidermal growth factor receptor 2 gene (ErbB2) signaling pathway in cardiomyocytes but also found that blocking Rho A during heart regeneration (or mitotically induced heart stimulations) resulted in changes in neuregulin 1 (Nrg1), Vegfaa, or vitamin D, disrupting muscle regeneration
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6A), this response was absent when we used USP33 harboring a mutated T154 residue, which is a known consensus Akt phosphorylation site (Fig
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