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Alternatively, the activation of receptor tyrosine kinase can inactivate GSK-3 via phosphorylation of upstream kinase Akt or extracellular signal regulated kinase (ERK), thereby allowing cytosolic -catenin to escape ubiquitin-dependent degradation [26]

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Pelleted Sf9 cells were lysed in lysis buffer containing 20 mM Tris pH7.5, 2.0 mg ml 1 iodoacetamide, 0.2 g ml 1 leupeptin (Sigma), 100 g ml 1 benzamidine (Sigma), and 0.5 M finasteride by stirring at 4 C for 1 h

[DOI] [PubMed] [Google Scholar] 139.Guo C., Wei J., Yang J., Xu J., Pang W., Jiang Y