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Description
When glutamate release exceeds glutamate uptake, the excess glutamate activates a large number of postsynaptic NMDA-Rs, resulting in the induction of excitotoxic neuronal death by allowing excessive Ca 2+ influx through the receptor-operated cation channels [73]
eLife 7, e38194
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This effect reflects their natural physiological role in the body
[DOI] [Google Scholar] Vzquez-Romero A
