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A recent study used a comprehensive approach to investigate the association between UA levels and neurodegenerative outcomes

Yeast Vps13 promotes mitochondrial function and is localized at membrane contact sites

Because typical early atherosclerotic lesion formation observed in hyperhomocysteinemic animals can be prevented by an antiplatelet therapy with dipyridamole, the authors concluded that platelet-mediated proliferation of smooth muscle cells is a potential cellular mechanism involved in the atherogenicity of hyperhomocysteinemia