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The FDAs stated concerns include the potential for immune reactions when a synthetic peptide is injected, difficulties in verifying the purity and composition of the compound, and insufficient safety data for the routes of administration being used [15]

The GPR30 receptor agonist G1 inhibits ferroptosis and significantly improves chondrocyte viability and motor ability in mice through the YAP1/FTH1 pathway, while GPR30 receptor antagonist G15 has the opposite effect, suggesting that hormone-related ferroptosis may emerge as a promising target for alleviating osteoarthritis in postmenopausal women

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