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Neuroprotective effects of CAQK in TBI To test CAQK for possible effects on brain injury, mice with CCI were treated with repeated i.v

Despite advances in surgical and pharmacological interventions, the persistence of such symptoms diminishes quality of life and daily functioning of patients ( A key pathological mechanism implicated in SCI and DCM is neuroinflammation driven by prolonged activation of microglia and macrophages (M/M) in the central nervous system ( Knowledge of the distribution of M/M in brain-spinal cord lesions in the chronic phase, including differences between those in SCI and DCM, is crucial for comprehending the underlying mechanisms and developing new treatment approaches for NeP in patients with SCI and DCM

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Protocol: Inject BPC-157 subcutaneously near the injury site

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