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Generally, YTHDF1 positively controls glycolysis through stabilizing transcripts or initiating translation in a wide range of cancers

Targeting lactate transporters (MCT1/4 inhibitors) has been shown to restore T cell glycolysis and reinvigorate ICI responsiveness

GlcN-6-P is acetylated to N-acetylglucosamine-6-P, which is then converted to N-acetylglucosamine-1-P by a mutase reaction, and it reacts with UTP to form UDP-n acetylglucosamine in the end, which is the final product of the pathway, providing substrates for post-translational modifications of the nucleus, cytoplasm, and mitochondrial proteins (57)

Xiong M, He Q, Lai H, Wang J

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