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It is also non-invasive, which reduces the likelihood of complications typically associated with more aggressive treatments

Other studies have revealed signs of oxidative stress without detecting oxidative damage

Neuro-inflammation, amyloid-beta plaques, mitochondrial damage, and increased oxidative stress contribute to cognitive decline

This connection is most likely related to vascular damage induced by homocysteine

Different exotoxins use a wide variety of strategies to traverse the intestinal barrier via complex interactions with the enterocytes or their surface receptors or even the protein complex that controls the opening size of the paracellular pathways [84, 85]