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Current evidence suggests that inflammation contributes to EDC through upregulation of central inflammatory cytokines, overactivation of microglia and astrocytes, and hyperactivation of the HPA axis, leading to neurotransmitter imbalance, impaired synaptic plasticity, increased pyroptosis, and OSpositioning EDC as a prototypical inflammation-driven and stress-driven neuropsychiatric comorbidity (Xu et al., 2018
Saunders NR, Liddelow SA, Dziegielewska KM (2012) Barrier mechanisms in the developing brain
Levi M, Thachil J, Iba T, Levy JH
Brain Pathol 25: 3543, 2015
Haythorne, E
