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The entry of cisplatin into the inner ear leads to the formation of inner ear cellular damage products (DAMPs), which, together with cisplatin, bind to toll-like receptors (TLRs) (Babolmorad et al., 2021) and activate downstream signaling, producing transcription factors (e.g., nuclear factor kappa B (NF-B), activator protein-1 (AP-1), and interferon regulatory factor 3 (IRF3)), which in turn regulate the expression and secretion of proinflammatory signaling molecules

Estimation of the Long-Term Cardiovascular Events Using UKPDS Risk Engine in Metabolic Syndrome Patients

[DOI] [PMC free article] [PubMed] [Google Scholar] 47.Sassi Y, et al

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