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The TrxRs differ from GR by having a catalytic selenocysteine redox-active site at the C-terminus in addition to the standard CXXC domain (Lu and Holmgren, 2014)

This disequilibrium can result in cellular injury and is implicated in the pathogenesis of neuropathy, wherein nerve damage arises from sustained OS

Methods Online databases, such as Scopus, Google Scholar, Web of Science, and PubMed/MEDLINE were systematically searched until December 2020 using keywords, such as review, neurological disorders, and CoQ10

10.3390/molecules28072982 123 RungratanawanichW.LinY.WangX.KawamotoT.ChidambaramS

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