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This result also confirms that selenium can inhibit the tumorigenesis caused by chemical carcinogens mainly in the initiation and promotion stages of chemical carcinogenesis, and its related mechanism may be related to the fact that selenium reduces the activity of hydroxylase that can activate carcinogens such as aromatic amine hydroxylase in the early stage of carcinogenesis, and increases the activity of glucuronosyltransferase that can release the toxicity of carcinogens, thus blocking the activation of the metabolism of carcinogens in the organism

PPC works differently: it physically disrupts the phospholipid bilayer of the adipocyte itself, lysing the cell and eliminating it from the treated area

Ferritinophagy-mediated ferroptosis is involved in sepsis-induced cardiac injury

Polycystic ovary syndrome and 25-hydroxyvitamin D: A bidirectional two-sample Mendelian randomization study

Cross-linkers form covalent bonds between interacting proteins, preventing dissociation during the assay