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Mutual interaction between oxidative stress and endoplasmic reticulum stress in the pathogenesis of diseases specifically focusing on non-alcoholic fatty liver disease

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Thus, GPx-1-deficient neurons have enhanced cell death after exposure to Abeta, whereas ebselen or N-acetyl-L-cysteine treatment or overexpression of GPx-1 significantly attenuated Abeta-induced toxicity (200)

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