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doi: 10.1074/jbc.M703091200
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Therefore, it was hypothesized that miR-483 could be co-transcribed with IGF2 due to the lack of a promoter of its own and its overexpression could contribute to the pro-oncogenic effect attributed to IGF2 (100)
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identified that the body releases GHK at the site of an injury, proposing that it may be an early signal for skin repair
