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Targeting mitochondrial dysfunction in the treatment of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) - a clinical audit

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COX17p deficiency does not cause immediate lethality in early embryonic development, but as development progresses, impaired copper ion transport into mitochondria disrupts cellular energy supply, ultimately proving fatal (53)

As with other stresses, GSH is thought to act as a signaling molecule or as a growth regulator to improve extreme temperature stress tolerance in plants (Hasanuzzaman et al