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The Structural Basis of the Autoinhibition Mechanism of Glycogen Synthase Kinase 3 (GSK3): Molecular Modeling and Molecular Dynamics Simulation Studies. Journal of Biomolecular Structure and Dynamics 38: 17411750

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In the ApoE/ mouse model induced by high-fat diet, liver-specific overexpression of GCN5L can reduce the area of atherosclerotic plaques, and its effect is superior to that of statin drugs as monotherapy ( Additionally, studies show that GCN5L plays a key role in glucose metabolism by regulating the acetylation state of insulin receptor substrates (IRS), thereby affecting insulin signaling ( In the field of cancer therapy, GCN5L demonstrates bidirectional regulatory potential in clinical applications

High mass balance values were obtained for all nanocarrier groups, which indicates the good recovery of the nanoparticles from the permeability samples (Table S3) and minimal sequestration in the cells or binding to the plastic surfaces of the culture-ware