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Activation of PLC- in these cells induces the phosphorylation of PKC-, leading to the translocation of NF-B and Jun N-terminal protein kinase (JNK) inside the nucleus, resulting in increased production of inflammatory cytokines ( Figure 4 )

It is highly expressed in the heart, muscle, and kidney and operates a sodium-dependent transport of carnitine and a sodium-independent organic cation transport [3]

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Apolipoproteins E and CIII interact to regulate HDL metabolism and coronary heart disease risk

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