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Sequencing was performed on an Illumina NovaSeq (S4) 300-cycle

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298 Under excessive ROS levels, ATM triggers downstream sites such as p53 and checkpoint kinase 2 (CHK2) for activation even when the DNA damage response (DDR) is absent, indicating that the activation pathway has no impact on the role of ATM at a certain level, whereas ATM has many other functions independent of the DDR 297 (Table 1, Fig

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Furthermore, NaBPC157 has selective effects on digitalis-induced cardiotoxicity

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