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Next, we quenched cellular NO in erlotinib-resistant cells with the NO-trap carboxy-PTIO and revealed that this partially re-sensitised PC9ER cells to erlotinib (Figure 5c)

PMID 15500036

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Ipamorelin produced a selective and dose-dependent increase in GH levels with no significant stimulation of ACTH or cortisol, suggesting hypothalamic specificity. Kraemer et al., Journal of Clinical Endocrinology & Metabolism 2

and regulation of gene expression via interaction with transcription factors such as STAT3 and Nrf2 24