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In selected contexts, modulate ACSL4/LPCAT3 (PUFA loading into membranes) and LOX activity to reduce peroxidation substrates and enzymatic initiation, while reinforcing parallel anti-ferroptotic circuitsFSP1CoQ at the plasma membrane and DHODHCoQH 2 in mitochondriato buffer lipid radicals without abolishing physiological signalling (Mao et al

Alteration of angiogenesis in Helicobacter heilmannii-induced mucosa-associated lymphoid tissue lymphoma: interaction with c-Met and hepatocyte growth factor

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Abstract Ferroptosis is an iron-dependent form of non-apoptotic cell death characterized by excessive lipid peroxidation and associated with a plethora of pathological conditions in the liver
