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Patients who develop symptoms of unexplained hyperammonemic encephalopathy while receiving valproate therapy should receive prompt treatment (including discontinuation of valproate therapy) and be evaluated for underlying urea cycle disorders (see Contraindications and Hyperammonemia and Encephalopathy Associated with Concomitant Topiramate Use as follows)

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Together, these mechanisms create comprehensive research tools for investigating fundamental questions about musculoskeletal tissue engineering, biomechanical property optimization, and regenerative capacity in laboratory models of tendon, ligament, muscle, and bone repair