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These functional declines are compounded by dysfunctional signaling in TLR2 and TLR4 pathways, further impairing immune responses in the elderly [259]

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The installation and development of these diseases are closely linked to metabolic changes that generate a state of oxidative stress, due to the excessive production of reactive oxygen species (ROS) and reactive nitrogen species (RNS) [7,8]

In models of Mycobacterium infection, macrophages with elevated HIF-1 expression demonstrate enhanced phagocytic and bactericidal activity (Zenk et al., 2021)