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Although the exact molecular mechanisms behind this relationship remain to be elucidated, multiple studies have suggested that dietary TMAO promotes atherosclerosiswhich can lead to coronary artery disease if left uncheckedvia a combination of poor metabolic control (specifically, glucose intolerance and insulin resistance), overexpression of inflammatory markers 1 , and altered bile acid metabolism 10

Adam et al., University of Washington, Seattle, 1993

These issues contribute to the limitations of the currentlyavailable literature to guide recommendations regarding management of persons with acute hepatitis C, a literature that consists of studies of uncontrolled case series receiving a variety of treatment regimens administered at varying times after acute infection

Apoe -/- mice lacking CD36 are more vulnerable to some bacterial infections (344) but also have less atherosclerosis when fed a high cholesterol diet (345)