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It represses autophagy in degenerative and metabolic disorders by destabilizing or activating inhibitory transcripts while promoting autophagy in cancers by stabilizing key ATG mRNAs or enhancing mitophagy pathways

ROS oxidize -SH groups of cysteine residues in protein kinases A, C, and D, receptor tyrosine kinase (RTK), and Ca/calmodulin independent protein kinase II, which in turn phosphorylate target proteins participating in various signaling mechanisms (Kruk et al

indicate, the ability of D + Q to eliminate senescent adipose-derived stem cells (ADSCs) could be beneficial in limiting ovarian cancer metastasis to the peritoneum and adipose tissue [117]

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doi: 10.3389/fmicb.2015.01543