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While determining causal relationships between these changes and embryological outcomes is objectively difficult, many sperm epigenetic changes have been associated with sperm abnormalities [115]

Our study demonstrated that inactivation of RIPK1 kinase through the introduction of the Ripk1 D138N mutation in hepatocellular HK2-deficient mice significantly ameliorated liver pathology by preventing hepatocyte apoptosis and subsequently reducing the development of HCC