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In this review, mechanisms of antiestrogen resistance will be introduced in the context of the presence of ER protein and function, genomic and nongenomic ER-mediated signaling network, pharmacologic and immunologic aspects, cell fates, cancer stem cells, and transcription factor-ER interactions

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Increased energy and fat metabolism allow patients to easily engage in physical activities, take balanced food, and observe improvement

Furthermore, matrix metalloproteinases (MMPs) are enzymes that degrade protein molecules in ECM and crucial molecules in the pathophysiologic process of disruption of BBB

CHCHD2 mutant mice display mitochondrial protein accumulation and disrupted energy metabolism