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In order for vitamin B12 to be absorbed, it binds with an important protein called intrinsic factor

Nerve Regeneration Peripheral nerve injury studies showed: Improved functional recovery Enhanced axonal regeneration Reference: Gwyer et al., 2019 5

Deficiency of Cbl, whether nutritional or due to inborn errors of Cbl metabolism, inactivate MS and MCM leading to the accumulation of homocysteine (Hcy) and methylmalonic acid (MMA), respectively

[DOI] [PMC free article] [PubMed] [Google Scholar] 14.Butler C.C., Vidal-Alaball J., Cannings-John R., McCaddon A., Hood K., Papaioannou A., Mcdowell I., Goringe A

While observational and mechanistic studies suggest that disturbed one-carbon metabolism, elevated homocysteine, and mitochondrial dysfunction may contribute to neurodegenerative pathways, current evidence does not establish a direct causal link between B12 deficiency and Alzheimers disease