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hence, when exogenous L-arginine was supplemented, a shift from glycolysis to OXPHOS was observed in cultured cells (34, 114)

While several subtype-specific therapeutic approaches have been identified for SCLC 36,37,38,39,40,41 , recent evidence suggests that SCLC subtype intratumoral heterogeneity may evolve as a consequence of subtype plasticity 5 highlighting the need to devise therapies targeting SCLC plasticity rather than isolated molecular subtypes

doi: 10.1027/1016-9040.14.2.168 56

doi: 10.3390/molecules191220091

Its mechanism is not only related to activating the Nrf2 signaling pathway and increasing the activity of antioxidant defenses but also to inhibiting the NF-B signaling pathway, which is closely related to reducing the release of proinflammatory factors [57]