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Neurotoxicity due to such increased DA is the outcome of reactive oxygen species (ROS) and oxidative products of DA.

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Cellular redox status presents broad implications with diverse physiological and pathological processes

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Mucosal samples were taken to evaluate the temporal relationship between the extent of injury, the levels of glutathione (GSH) during acute colitis induced by TNBS+ethanol, and the effect of N-acetylcysteine (NAC) administration

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