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One possibility is that pink-1 and pdr-1 operate independently to regulate -synuclein toxicity in worm dopaminergic neurons and loss of each gene is responsible for an additive phenotype observed in double mutants
Or are you simply responding to health trends
Z.et al (2021)
See frequency by goal (skin, hair, healing) and cycling schedules
Sep-Oct 2009;75(5):701-5
