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has reported that administration of IKK inhibitors BAY-65-1942 inhibited the microglial activation, attenuated the loss of dopamine neurons, and enhanced levels of neurotransmitters [71], and the similar result was observed after administration of IKK inhibitors, such as BAY-65-1942, metformin, and ginsenoside Rb1, and peptide [70, 74, 75]

Several prognostic risk scores have been developed to assess disease severity, mortality, and pharmacologic intervention in patients with AAH

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