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[DOI] [PMC free article] [PubMed] [Google Scholar] 136.Tang W., Levin L., Talaska G., Cheung Y.Y., Herbstman J., Tang D., Miller R.L., Perera F., Ho S.M

Specifically, oxidative stress induced by HBV activates PRKAA (the catalytic subunit of AMPK), enhancing autolysosome-dependent degradation by increasing cellular ATP levels, which depletes autophagic vacuoles [191]

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