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These controlled in-vitro models are absolutely crucial for understanding age-related metabolic decline and evaluating potential interventions for lipid storage dysfunctions

The c-Jun NH 2 -terminal kinase (JNK) pathway is activated by OS and cytokines (tumor necrosis factor- and FAS) and, similar to the ERK pathway, stimulates a cascade of phosphorylation reactions involving MAPKKK, MAPKK, and ultimately phosphorylating JNK on threonine and tyrosine residues

doi: 10.2174/138920011796504536 9 PoorabbasABaddadchiJFallahFMahdaviRAliasgarzadehAAsadiYK

Monitoring of estrous cycle in the laboratory rodent by vaginal lavage