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Wright et al., 2012), which clearly demonstrate that both the peripheral and central nervous system actions of AT 4 receptor antagonists depend on their ability to inhibit the hepatocyte growth factor (HGF)/c-Met (HGF receptor) system by binding to and blocking HGF activation

These changes can cause oxidative modifications to reactive thiols of structurally- or functionally-critical proteins, such as transcription factors 43 , signal transduction molecules 44 or metabolic enzymes 45 , and thereby evoke cellular defense mechanisms against oxidative damage 46

The evolution of dichloroacetate (DCA) as a pharmaceutical agent has a more convoluted history (202)

Reference: Jiang J, Wu D, Wang Y, et al

doi: 10.1016/s0022-5347(17)39009-2