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Preclinical models have shown the efficacy of AAV5-GBA and AAV9-GBA in preventing dopamine neuron loss (Rocha et al

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Possible mechanisms for the deleterious effects of Hcy on cognitive function are as follows: Hcy leads to DNA breakdown, oxidative damage, and the apoptotic process [7], in addition to as well as excitotoxic cell death by directly activating the neuronal NMDA receptor after formation of homocysteic acid [45]

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HOX antisense lincRNA HOXA-AS2 promotes tumorigenesis of hepatocellular carcinoma