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Pro-inflammatory cytokines such as TNF-, IL-1, and IL-6 contribute to microglial activation in the auditory brainstem and cortex, exacerbating neural dysregulation
It may be necessary to attack each of these likely causes with separate drug development programs
As there is no protein turnover or repair within the anucleate fibre cells, post translational modifications (such as glycation) to antioxidant enzymes would undoubtedly lead to altered function over time
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