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Suppression of NFS1 alone does not induce ferroptosis, but when cells produce large amounts of ROS, iron starvation induced by inhibition of NFS1 promotes ferroptosis

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Through these combined effects, ApoE4 promotes mitochondrial instability and defective mitochondrial turnover, ultimately exacerbating neuronal vulnerability and disease progression (Chen H

[DOI] [PMC free article] [PubMed] [Google Scholar] 63.Egner P.A., Chen J.G., Wang J.B., Wu Y., Sun Y., Lu J.H., Zhu J., Zhang Y.H., Chen Y.S., Friesen M.D., et al

Gill SS, Tuteja N