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doi: 10.3389/fneur.2023.1100933 169 ObengE

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Low-molecular-weight thiols in thioldisulfide exchange

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Lipid peroxidation increases ROS generation and damages cell membranes, activating a series of pro-apoptotic signaling molecules (e.g., caspase-3, p53), which lead to muscle cell apoptosis

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U54HD061221), which is a part of the National Institutes of Health (NIH) Rare Disease Clinical Research Network (RDCRN), supported through collaboration between the Office of Rare Diseases Research (ORDR), the National Center for Advancing Translational Science (NCATS) and the Eunice Kennedy Shriver National Institute of Child Health and Human Development (NICHD), and NIH K08DK106453

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At present, VEGF-A and VEGF-C have emerged as the extensively studied soluble factors associated with the development of a pre-metastatic microenvironment within the SLN (193, 194)

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