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These references make clear that B12 therapy is primarily about treating deficiency, not directly burning fat
NO generated by iNOS could interact with lipid peroxides and secondary lipid radical intermediates catalyzed by 15-LOX (70) and substitute GPX4 as an anti-ferroptotic defense, which renders M1 macrophages with high resistance to pharmacologically induced ferroptosis
doi:10.1016/j.clnesp.2017.01.002
and Buettner G.R
doi: 10.1016/j.freeradbiomed.2021.09.008 219 ZengLYangKYuGHaoWZhuXGeAet al
