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Distinct stressors induce stress-related modulations that represent a conventional yet conceptually novel form of plasticity, which may have been both inherited and acquired through evolution and selection

In summary, these findings reveal that TGF1 downregulates DLD-K430ac through the SMAD3/4-dependent mitochondrial-nuclear translocation of ACAT1

Similarly peptide-drug conjugates can be designed for targeting EGFR in TNBC

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