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Research began on cholesterol-driven changes and revealed that IMC treatment increases fecal neutral sterol loss and reduces intestinal sterol transporter NPC1L1 expression, thereby preventing diet-driven hepatic cholesterol accumulation and altering bile acid metabolism

January 2012;7(1):131138

Whereas the dose plays a role, the 7-day half-life of T 4 allows physiologic compensation, such as the suppression of T 4 to T 3 conversion, inhibition of endogenous hormone production by negative feedback loops, and downregulation of thyroid hormone receptors

Nitric oxide, reactive oxygen species, and skeletal muscle contraction

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