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Description
L., Malka, O., Hefetz, A., Tarpy, D
In the context of MASLD, glucagon receptor agonism may complement GLP-1 receptor agonism by directly stimulating hepatic fatty acid oxidation and reducing lipogenesis, leading to decreased hepatic steatosis
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While helpful, they face a problem: absorption limitations
Below, we summarize novel therapeutic targets and strategies with clinical application potential to provide more options for disease treatment (Table 1)
