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Replacement of Tyr110 in recombinant Y110A decreased both k cat app and 1/ values for the disulfide substrate and GSH in the GSSCys and HEDS assay (Supplementary Figs 18 and 19)

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Although the mechanisms destructive to pancreatic acinar cells are unknown, once a cellular injury pattern has been initiated, activated neutrophils exacerbate the problem by releasing superoxide (the respiratory burst) or proteolytic enzymes (cathepsins B, D, and G, collagenase and elastase)

This study was supported by Liaoning Provincial Science and Technology Plan Project (2020JH2/10300118)

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