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The most actionable approach is tracking the downstream markers that respond to glutathione status: oxidative stress markers, liver enzymes, methylation markers, and inflammation
One of the key players in the NEM stress response is the release of stress hormones like cortisol via the adrenal glands
Most predictive of poor clinical outcomes at 3 and 12 months were increases in LPCs (14:0, 16:0, 16:1, 17:0, 18:0, 18:2, 20:3 and 28:1), ketogenic AA, acylcarnitines, as well as BCAAs, NAA (n-acetylaspartate upregulates lipid degradation and fatty acid mobilization [48]), and tyrosine over the first four days after injury
doi: 10.2337/diabetes.49.9.1543
What human clinical trial data exists to inform dosing and safety expectations
