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Description
During processes such as halogenation, nitration, and the formation of protein cross-links through tyrosyl radicals, eosinophil peroxidase, and MPO contribute to oxidative stress
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doi: 10.1016/s0264-410x(99)00214-5

Rather than directly abrogating LDS-mediated interactions, phosphorylation of GABARAPL2 at S10 by TBK1 selectively disrupts the IRGQ-GABARAPL2 interface of these scaffold complexes, indirectly destabilizing the assembly to reduce the overall recruitment efficiency of ATG8-associated machinery

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