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Consistent with this role, modulation in glycinergic signaling has been related to benefits against symptoms in a broad spectrum of neurological and psychiatric conditions, including epilepsy, schizophrenia, cerebral ischemia, and neurodegenerative disorders

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Under normal circumstances, NRF2 forms a complex with its inhibitory protein, Keap1 which regulates the stability and nuclear translocation of NRF2, thereby controlling its activity

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These inhibitors induce ferroptosis by blocking cystine uptake via SLC7A11 and fall under the category of class I ferroptosis inducers