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Description
The molecular docking data suggest that the LCS3 nitro subgroup displaces the His119 His126 hydrogen bond between subunits

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By acknowledging the active role of glial cells, securing robust biomarkers, and designing multimodal, personalized strategies, the field can move closer to genuine disease modification

DJ-1 is a redox-dependent molecular chaperone that inhibits alph-synuclein aggregate formation
