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Funding The authors would like to acknowledge funding from the Research Start-up Fund of Jining Medical University (Reference: 600791001), the Foundation of Medical Health Science and Technology Development Program of Shandong Province (Reference: 202002061311), the Research Support Fund for Teachers of Jining Medical University (Reference: JYFC2019KJ013), and the Jining Medical College Student Innovation and Entrepreneurship Training Program Project (Reference: cx2021053)

Intracellular stores of Ca 2+ regulate cellular redox equilibrium by ROS formation and elimination, allowing cells to shift the redox environment towards either a more oxidizing or reducing state

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