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discovered that inhibition of histone deacetylase might alleviate abnormalities in the Notch and Eph axis in prion protein PrP deficient and prion-infected cells (87)

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A study of LGR-7, a primary receptor of RLX-2, has shown that RLX-2 suppressed collagen-I, PAI-1, and IL-8 expression in ESCs [82] and all these molecules have an important role in fibrosis formation [83, 84]

doi: 10.1002/prot.10559

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