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Interestingly, in human subjects, as well as in animal models, there appears to be a limited range for the beneficial effects of selenium: both too much and too little may have detrimental effects on health (reviewed in [21, 22])

[12] Damage to the liver is not due to the drug itself but to a toxic metabolite ( N -acetyl- p -benzoquinone imine (NAPQI)) produced by cytochrome P-450 enzymes in the liver

Metabol Open (2021) 10:100093

After endocytosis, the drugs are released to precisely destroy AML cells, while normal cells are spared due to their low expression of target antigens (136)

These models bridge cellular mechanisms to systemic toxicity assessments [176]